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For example, a normally functioning body already produces collagen and GHRH, so adding collagen peptides or a GHRH analog should only enhance naturally occurring biological processes

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Intriguingly, because cytoplasmic condensation is not seen in all dead cells, our results suggest that alternative cell death pathways—which may involve cellular damage to nucleic acids and proteins resulting from both the primary drug–target interaction and subsequent generation of reactive metabolic by-products—also contribute to antibiotic-induced cell death