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The patient’s presentation aligns well with reported literature on hyperthyroidism-induced hepatic injury, particularly the combination of elevated liver enzymes, severe hyperbilirubinemia, and profound metabolic derangements ( Firstly, the patient’s autonomous discontinuation of antithyroid medications complicates the clinical picture
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The compound binds to growth hormone receptors and triggers fibroblast proliferation, accelerating collagen production at injury sites
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