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Male C57Bl6 mice subjected to experimental myocardial infarction and treated with the CXCR4 inhibitor AMD3100 (1.25 mg/kg/d), in combination with diprotin A (70 mg/kg twice daily) and G-CSF (100 μg/kg/d), demonstrated an increase in infarct size 30 days after myocardial infarction, suggesting that the beneficial effects observed were mediated directly through a SDF-1α/CXCR4 signaling axis (186)

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doi: 10.1016/j.expneurol.2008.12.002